(ZIP 3 MB) Additional file 7: Table S7 Statistically significant

(ZIP 3 MB) Additional file 7: Table S7. Statistically significantly

differentially expressed probe sets in the gingival tissues according to levels of P. micra in the adjacent pockets. (ZIP 3 MB) Additional file 8: Table S8. Statistically significantly differentially expressed probe sets in the gingival tissues according to levels of C. rectus in the adjacent pockets. (ZIP 3 MB) Additional file 9: Table S9. Statistically significantly differentially expressed probe sets in the gingival tissues according to levels of E. corrodens in the adjacent pockets. buy Panobinostat (ZIP 3 MB) Additional file 10: Table S10. Statistically significantly differentially expressed probe sets in the gingival tissues according to levels of V. parvula in the adjacent pockets. (ZIP 3 MB) Additional file 11: Table S11. Statistically significantly differentially expressed probe sets in the gingival tissues according to levels of A. naeslundii in the adjacent pockets. (ZIP 3 MB) Additional file 12: Table S12. A list of the top 100 differentially expressed probe sets in the gingival tissues according to levels of ‘Etiologic burden’ in the adjacent pockets. (XLS 32 KB) Additional file 13: Table S13.

A list of the top 100 differentially expressed probe sets in the gingival see more tissues according to levels of ‘Putative burden’ in the adjacent pockets. (XLS 26 KB) Additional file 14: Table S14. A list of the top 100 differentially expressed probesets in the gingival tissues according to levels of ‘Health-associated burden’ in the adjacent pockets. (XLSX 17 KB) Additional file 15: Table S15. List of all statistically significantly regulated GO groups in the gingival tissues according to levels of each of the 11 investigated species in the adjacent pockets. (ZIP 646 KB) References 1. Socransky SS, Haffajee AD: Periodontal microbial ecology. Periodontol 2000 2005, 38:135–187.CrossRefPubMed 2. Marsh PD: Dental plaque: biological significance of a biofilm and community lifestyle. J Clin Periodontol 2005,32(Suppl 6):7–15.CrossRefPubMed 3. Listgarten MA, Helldén Thalidomide L: Relative distribution of bacteria at clinically healthy and periodontally diseased sites in humans. J Clin Periodontol

1978,5(2):115–132.CrossRefPubMed 4. Socransky SS, Haffajee AD, Smith C, Dibart S: Relation of counts of microbial species to clinical status at the sampled site. J Clin Periodontol 1991,18(10):766–775.CrossRefPubMed 5. Page RC, Schroeder HE: Pathogenesis of inflammatory periodontal disease. A summary of current work. Laboratory investigation; a journal of technical methods and pathology 1976,34(3):235–249.PubMed 6. Offenbacher S: Periodontal diseases: pathogenesis. Ann Periodontol 1996,1(1):821–878.CrossRefPubMed 7. Chung CH, Bernard PS, Perou CM: Molecular portraits and the family tree of cancer. Nat Genet 2002,32(Suppl):533–540.CrossRefPubMed 8. Quackenbush J: Microarray analysis and tumor classification. N Engl J Med 2006,354(23):2463–2472.CrossRefPubMed 9.

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