CdS/Si-NPA was obtained
by depositing a continuous film of CdS onto silicon nanoporous pillar array (Si-NPA) via a chemical bath deposition (CBD) method and the average reflectance of CdS/Si-NPA was less than 7% in the wavelength range of 200-1000 nm. Under 1 sun air mass (AM) 1.5 G illumination, CdS/Si-NPA exhibits an obvious photovoltaic effect. These results indicate that CdS/Si-NPA has a strong broadband optical antireflection and might be a promising candidate for the assembly of high efficiency solar cells. (C) 2011 American Institute of Physics. [doi:10.1063/1.3658814]“
“A 64-yr-old man with end-stage kidney disease caused by hypertensive nephrosclerosis underwent living-unrelated ABO-identical kidney transplantation (KTx) at the age of 60 yr from his 60-yr-old wife. Maintenance trough concentration Selleck Alvocidib of cyclosporine A (CsA) was 100 +/- 30 ng/mL. Five months after KTx, proteinuria gradually increased to around Pevonedistat 1 g/d. TRBx at eight months after KTx revealed the new-onset alteration of mild arteriolosclerosis with intimal hyalinosis, which might reflect calcineurin inhibitor (CNI)associated arteriopathy (CAA). Nearly one and half years after KTx, urinary protein excretion
became nearly 2 g/d. TRBx revealed the advanced CAA and findings of focal segmental glomerulosclerosis (FSGS). Then, CNI was switched from CsA to tacrolimus (TAC). TRBx at two and half years after KTx revealed progressed arteriolar transmural thickening and striped fibrosis, which were supposed to be induced by an increase in
serum TAC concentration because of acute enterocolitis. Then, TAC dose was reduced to serum trough concentration 58 ng/mL, but urinary protein excretion was increased up to 10 g/d. Reduction of TAC to trough concentration 2.0 +/- 0.5 ng/mL reduced urinary protein excretion. Attempts to elevate TAC trough concentration within normal range (48 ng/mL) reproducibly induced the recurrence of an increase in sCr or urinary protein excretion. All these findings supported the etiology of Nirogacestat graft dysfunction, and proteinuria of this case was FSGS.”
“The aim of this study was to determine if a meloxicam hydrogel could be administered in vivo via phonophoretic transdermal delivery using pulsed ultrasound by examining its anti-hyperalgesic effects in a rat carrageenan inflammation model. Carrageenan (1%) was injected into the plantar surface of the right hindpaw, and meloxicam hydrogel was administered via phonophoretic transdermal delivery. Changes in the mechanical and thermal hyperalgesia, as well as swelling, showed that phonophoretic delivery of meloxicam exhibited significantly better anti-hyperalgesic and anti-inflammatory effects than pulsed ultrasound. Topical and systemic application of meloxicam hydrogel using phonophoresis showed similar anti-hyperalgesic effects.